MyoTosterone a 38% Testosterone Increase – Legit?  Or not?

Testosterone boosters are typically sold as a culmination of a bunch of herbs that have shown promise in rats, particularly for increasing their mating behavior. In general this has translated very poorly to human males. That is why supplement studies, from American entities (or other scientifically equivocal countries) on healthy males often get significant media attention.

The latest study was funded and completed by Triarco Industries, which is most well know for their product Aminogen (found in many whey proteins).  The product they put to the test is called MyTosterone and contains both astaxathin extracted from Haematococcus algae (pluvialis), and saw palmetto.

The study itself used untrained men and included a huge age range between 20 and 68 years old (with no mention as to why).  There were three test groups including an 800 mg per day supplemented group, a 1200 mg per day supplemented group, and a placebo group. The administration period lasted 14 days (same length as the first popularized study with D-Aspartic Acid).

They found that after 14 days the 800 mg per day group had no significant increase in testosterone.  But the 1200 mg per day group had a 38% increase in testosterone.  Unfortunately a major weakness of this study was it’s lack of data reporting.  They did not list the individual baseline testosterone levels in the men.  That’s not too disappointing, but in this case they didnt even give the before and after average testosterone levels for each group.  And as you can see in the following table, the 38% increase vs. placebo is largely because the placebo group had a fall in testosterone over the 14 days.

astaxanthin boosts testosterone

Overall this seems very suspect, and here’s why…

Triarco Industries and ConsumberLab.com

ConsumerLab.com claims to be an independent third party tester of dietary supplements.  Meaning the information they report should be fully unbiased (unless perhaps they are paid to test a supplement by the company that makes it).   Per ConsumerLab.com:

consumerlab.com

Oddly enough the author (and only listed contributor) was Mark Anderson, whose email is at (@) consumerlab.com.  He also apparently works for Triarco Industries (or use to per wikipedia?  Hard to say…).  Nonetheless we will see if the next consumerlab.com report suggests MyoTosterone as the next greatest testosterone boosting supplement.  Anyways, we will see how the supplement industry spins this lackluster study into something it’s not, which is a decent testosterone booster.


 

One thought on “Astaxanthin Boosts Testosterone 38%

  1. DeGraff says:

    Astaxanthin lowers plasma TAG concentrations and increases hepatic antioxidant gene expression in diet-induced obesity mice.

    Br J Nutr. 2014 Oct 20;:1-8

    Authors: Yang Y, Pham TX, Wegner CJ, Kim B, Ku CS, Park YK, Lee JY

    Abstract
    Non-alcoholic fatty liver disease (NAFLD) is significantly associated with hyperlipidaemia and oxidative stress. We have previously reported that astaxanthin (ASTX), a xanthophyll carotenoid, lowers plasma total cholesterol and TAG concentrations in apoE knockout mice. To investigate whether ASTX supplementation can prevent the development of NAFLD in obesity, male C57BL/6J mice (n 8 per group) were fed a high-fat diet (35 %, w/w) supplemented with 0, 0·003, 0·01 or 0·03 % of ASTX (w/w) for 12 weeks. The 0·03 % ASTX-supplemented group, but not the other groups, exhibited a significant decrease in plasma TAG concentrations, suggesting that ASTX at a 0·03 % supplementation dosage exerts a hypotriacylglycerolaemic effect. Although there was an increase in the mRNA expression of fatty acid synthase and diglyceride acyltransferase 2, the mRNA levels of acyl-CoA oxidase 1, a critical enzyme in peroxisomal fatty acid β-oxidation, exhibited an increase in the 0·03 % ASTX-supplemented group. There was a decrease in plasma alanine transaminase (ALT) and aspartate transaminase (AST) concentrations in the 0·03 % ASTX-supplemented group. There was a significant increase in the hepatic mRNA expression of nuclear factor erythroid 2-related factor 2 and its downstream genes, which are critical for endogenous antioxidant mechanism, in the 0·03 % ASTX-supplemented group. Furthermore, there was a significant decrease in the mRNA abundance of IL-6 in the primary splenocytes isolated from the 0·03 % ASTX-supplemented group upon lipopolysaccharide (LPS) stimulation when compared with that in the splenocytes isolated from the control group. In conclusion, ASTX supplementation lowered the plasma concentrations of TAG, ALT and AST, increased the hepatic expression of endogenous antioxidant genes, and rendered splenocytes less sensitive to LPS stimulation. Therefore, ASTX may prevent obesity-associated metabolic disturbances and inflammation.

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